Cat. #158408
Anti-CUGBP2/ETR-3 [1H2-1F12]
Cat. #: 158408
Unit size: 100 ug
Target: CUGBP2/ETR-3 (Elav-like family member 2)
Class: Monoclonal
Application: WB ; IHC ; IF ; IP
Reactivity: Human ; Rat ; Mouse
Host: Mouse
£300.00
This fee is applicable only for non-profit organisations. If you are a for-profit organisation or a researcher working on commercially-sponsored academic research, you will need to contact our licensing team for a commercial use license.
Contributor
Institute: University of Florida
Tool Details
*FOR RESEARCH USE ONLY (for other uses, please contact the licensing team)
- Name: Anti-CUGBP2/ETR-3 [1H2-1F12]
- Alternate name: BRUNOL3, NAPOR; CELF2
- Research fields: Genetics;Tissue-specific biology
- Class: Monoclonal
- Conjugation: Unconjugated
- Molecular weight: 52
- Reactivity: Human ; Rat ; Mouse
- Host: Mouse
- Application: WB ; IHC ; IF ; IP
- Description: CUGBP2/ETR-3 is an RNA-binding protein containing 2 N-terminal and 1 C-terminal RNA recognition motifs. It has been shown to interact wtih A1CF. This protein promotes exon inclusion and its activities are required for appropriate splicing in cardiomyocytes. During development ETR-3 is expressed in the heart in both the nucleus and cytoplasm and its expression is down-regulated during heart development.
- Immunogen: Synthetic peptide corresponding to amino acids 209-415 of human CuGBP2
- Isotype: IgG1
Target Details
- Target: CUGBP2/ETR-3 (Elav-like family member 2)
- Molecular weight: 52
- Target background: CUGBP2/ETR-3 is an RNA-binding protein containing 2 N-terminal and 1 C-terminal RNA recognition motifs. It has been shown to interact wtih A1CF. This protein promotes exon inclusion and its activities are required for appropriate splicing in cardiomyocytes. During development ETR-3 is expressed in the heart in both the nucleus and cytoplasm and its expression is down-regulated during heart development.
Applications
- Application: WB ; IHC ; IF ; IP
Handling
- Format: Liquid
- Unit size: 100 ug
- Shipping conditions: Shipping at 4° C
References
- Ladd et al. 2005. Dev Dyn. 233(3):783-93. PMID: 15830352.
