MOZ fusion proteins block normal differentiation of myeloid progenitors resulting in AML (acute Myloid Leukaemia).Recombinant fragment of human MOZ (172-321aa) produced as a fusion protein in E. coli.
| Inventor | Institute |
|---|---|
| David Heery ; Tasneem Ritchie | University of Oxford |
| Cat. #: | 151838 |
|---|---|
| Tool sub type: | Primary antibody |
| Unit size: | 100 ug |
| Research Fields: | Genetics |
| Application: | IP ; WB ; IP ; WB |
| Target: | Monocytic Leukaemia Zinc Finger Protein, MOZ, MYST3 |
| Reactivity: | Human |
| Clone: | 127A |
| Host: | Mouse |
| Class: | Monoclonal |
| Product description: | MOZ fusion proteins block normal differentiation of myeloid progenitors resulting in AML (acute Myloid Leukaemia).Recombinant fragment of human MOZ (172-321aa) produced as a fusion protein in E. coli. |
|---|---|
| Conjugation: | Unconjugated |
| Isotype: | IgG3 kappa |
| Molecular weight: | 225 kDa |
| Immunogen: | GST-MOZ (172-321aa) recombinant protein human MOZ |
| Target background: | MOZ fusion proteins block normal differentiation of myeloid progenitors resulting in AML (acute Myloid Leukaemia).Recombinant fragment of human MOZ (172-321aa) produced as a fusion protein in E. coli. |
|---|
| Format: | Liquid |
|---|---|
| Concentration: | 1 mg/ml |
| Storage buffer: | PBS with 0.02% azide |
| Storage conditions: | -80° C |
| Shipping conditions: | Dry ice |
| References: |
Elizabeth Deeves. 2012. PhD thesis. Novel functions of the MOZ double PHD finder domain. |
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