#151029

Anti-HSVICP8 [11E2]

Cat. #151029

Anti-HSVICP8 [11E2]

Cat. #: 151029

Sub-type: Primary antibody

Unit size: 100 ug

Availability: 3-5 days

Target: Herpes Simplex Virus Infected cell protein 8 (HSV-ICP8)

Class: Monoclonal

Application: IHC ; IF ; IP ; WB

Reactivity: Virus

Host: Mouse

£300.00

This fee is applicable only for non-profit organisations. If you are a for-profit organisation or a researcher working on commercially-sponsored academic research, you will need to contact our licensing team for a commercial use license.

Contributor

Inventor: Roland Wolf

Institute: Cancer Research UK, London Research Institute: Clare Hall Laboratories

Tool Details
Target Details
Applications
Handling
References

Tool Details

*FOR RESEARCH USE ONLY (for other uses, please contact the licensing team)

  • Name: Anti-HSVICP8 [11E2]
  • Research fields: Microbiology
  • Clone: Array
  • Tool sub type: Primary antibody
  • Class: Monoclonal
  • Conjugation: Unconjugated
  • Molecular weight: 51 kDa
  • Strain: Balb/c
  • Reactivity: Virus
  • Host: Mouse
  • Application: IHC ; IF ; IP ; WB
  • Description: ICP8 is the major DNA binding protein of herpes simplex virus type 1.
  • Immunogen: ICP8 purified from U-35-VERO cells.
  • Isotype: IgG1

Target Details

  • Target: Herpes Simplex Virus Infected cell protein 8 (HSV-ICP8)
  • Molecular weight: 51 kDa
  • Target background: ICP8 is the major DNA binding protein of herpes simplex virus type 1.

Applications

  • Application: IHC ; IF ; IP ; WB

Handling

  • Format: Liquid
  • Concentration: 1 mg/ml
  • Unit size: 100 ug
  • Storage buffer: PBS with 0.02% azide
  • Storage conditions: -15° C to -25° C
  • Shipping conditions: Shipping at 4° C

References

  • Mohni et al. 2013. J Virol. 87(1):531-42. PMID: 23097436.
  • Efficient herpes simplex virus 1 replication requires cellular ATR pathway proteins.
  • Alazard-Dany et al. 2009. PLoS Pathog. 5(3):e1000340. PMID: 19282980.
  • Definition of herpes simplex virus type 1 helper activities for adeno-associated virus early replication events.